ADB-5'F-BUTINACA

From WikiMD's Food, Medicine & Wellness Encyclopedia

ADB-5'F-BUTINACA is a synthetic cannabinoid that has been identified in designer drugs. This compound is a derivative of BUTINACA, which is a compound originally developed by Pfizer as an analgesic medication. ADB-5'F-BUTINACA is a potent agonist of the CB1 receptor and CB2 receptor, which are part of the endocannabinoid system in the human body.

Chemistry[edit | edit source]

ADB-5'F-BUTINACA is a member of the cannabinoid class of chemicals. It is structurally related to other synthetic cannabinoids such as ADB-BUTINACA and 5F-ADB. The compound is characterized by a 1-amino-3,3-dimethyl-1-oxobutan-2-yl group, which is also present in its parent compound BUTINACA. The presence of a fluorine atom on the pentyl chain distinguishes ADB-5'F-BUTINACA from its analogs.

Pharmacology[edit | edit source]

As a synthetic cannabinoid, ADB-5'F-BUTINACA acts as a potent agonist for the CB1 and CB2 receptors. These receptors are part of the endocannabinoid system, which plays a crucial role in regulating a variety of physiological processes including pain sensation, mood, and memory. The activation of these receptors by ADB-5'F-BUTINACA can produce a range of effects, including analgesia, euphoria, and changes in perception.

Legal Status[edit | edit source]

The legal status of ADB-5'F-BUTINACA varies by country. In many jurisdictions, it is classified as a controlled substance due to its potential for abuse and the health risks associated with its use. In the United States, ADB-5'F-BUTINACA is a Schedule I controlled substance under the Controlled Substances Act.

Health Risks[edit | edit source]

The use of ADB-5'F-BUTINACA, like other synthetic cannabinoids, can pose significant health risks. These include acute toxic effects such as agitation, tachycardia, and seizures, as well as long-term effects such as dependence and cognitive impairment. The lack of quality control in the production of designer drugs also means that users may be exposed to other harmful substances.

See Also[edit | edit source]

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Contributors: Prab R. Tumpati, MD