Autistic catatonia

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Autistic Catatonia is a neurodevelopmental disorder characterized by a significant decrease in motor activity and communication in individuals with autism spectrum disorder (ASD). It is a complex and multifaceted condition that can manifest in a variety of ways, including mutism, stereotypy, negativism, and posturing.

Definition and Classification[edit | edit source]

Autistic catatonia is classified as a subtype of catatonia, a syndrome characterized by motor abnormalities such as akinesia (lack of movement), hypokinesia (reduced movement), and hyperkinesia (excessive movement). In the context of ASD, these motor abnormalities are often accompanied by significant impairments in social interaction and communication.

Symptoms and Signs[edit | edit source]

The symptoms of autistic catatonia can vary widely from person to person, but often include:

  • Mutism: A decrease or complete loss of speech.
  • Negativism: Resistance to instructions or attempts to be moved.
  • Posturing: Holding unusual body positions for extended periods of time.
  • Stereotypy: Repetitive, purposeless movements such as rocking or hand-flapping.

Causes[edit | edit source]

The exact cause of autistic catatonia is unknown, but it is thought to be related to abnormalities in the brain's neurotransmitter systems. Some researchers believe that it may be triggered by stress or trauma, while others suggest that it may be a result of underlying genetic factors.

Diagnosis[edit | edit source]

Diagnosis of autistic catatonia is based on clinical observation and assessment. The Bush-Francis Catatonia Rating Scale (BFCRS) is often used to measure the severity of symptoms.

Treatment[edit | edit source]

Treatment for autistic catatonia typically involves a combination of pharmacotherapy and behavioral therapy. Medications such as benzodiazepines and antipsychotics may be used to manage symptoms, while behavioral interventions aim to improve communication and social skills.

See Also[edit | edit source]

References[edit | edit source]

External Links[edit | edit source]


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Contributors: Prab R. Tumpati, MD